Abstract
Large sample sizes, high-resolution arrays and comprehensive imputation are pushing genetic fine-mapping of complex trait loci to its limits without, in most cases, pinpointing a unique variant-gene combination. Superimposing these results on sophisticated maps of functional chromatin elements promises to break this logjam, as a new study of type 2 diabetes compellingly demonstrates.
Original language | Undefined/Unknown |
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Pages (from-to) | 1373-1374 |
Number of pages | 2 |
Journal | Nature Genetics |
Volume | 47 |
Issue number | 12 |
DOIs | |
Publication status | Published - 2015 |
ASJC Scopus subject areas
- Genetics